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If the phenotypes of the mutants can be successfully restored to wild type, this will confirm that only the correct genes have been deleted and further validate the specific phenotypes ascribed to each GDH mutant

Erastin, the first ferroptosis inducer discovered, targets SLC7A11, leading to GSH depletion and subsequent GPX4 inactivation, leading to ferroptosis [8]

Furthermore, another recently described player involved in GSH catabolism is the specific cytoplasmic GSH-degrading enzyme CHAC1 (59)

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Conversely, ARA itself can activate key oxidative stress molecules

It has been shown that exercise causes MOTS-c levels to go up in humans